<span>Living things are biotic factors like a dog, fungi and trees
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Given what we know, we can confirm that the aspect of nucleic acids and transcription that can help explain this discrepancy is that of alternative splicing.
<h3>Alternative Splicing.</h3>
- This process allowed the cells to use a single gene for the creation of various proteins.
- This process includes the separation of RNA at splicing sites to create new versions of the mRNA strand.
- This helps to increase the diversity of mRNA's available and create more proteins.
Therefore, given that alternative splicing allows the cells to create multiple forms of distinct proteins from a single gene, this allowed our organisms to develop the vast number of proteins we use, without the need for a much larger genome.
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Answer:
Chromosomes can exchange genetic information during a process called "crossing over." This occurs when homologous chromosomes are lined up in pairs. This recombination of maternal and paternal genetic material is a key feature of meiosis.
Explanation:
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Answer: runoff is like when rain and water drains away
Explanation:
<span>Cells control cell division in order to maintain normal cell function. If something happens to the control of the cell division, the healthy cells will divide uncontrollably. These new cells are cancer cells. </span>
The mutations in three genes are responsible for development of cancers:
1. Mutation in proto-oncogenes. Proto-oncogenes normally signal cells to grow and differentiate. Proto-oncogenes can become oncogenes due to mutations which result in the uncontrollable division of the cells.
2. Mutation in tumor suppressor genes. In normal cells, tumor suppressor genes suppress genes essential for cell cycle and that way they prevent uncontrollable cell division. However, after a mutation in these genes, suppression is lost and the cell may progress to cancer.
3. Mutation in stability genes. In normal cells, they have no role either in cell death or growth, but they control mutation rate. Mutation in stability genes results in situation where all genes, including proto-oncogenes and tumor suppression genes, are more frequently mutated.