Answer: right bottom is kidney
Left bottom is liver
Q. 4 part a, The excretory system.
B, kidney: The kidneys make urine by filtering wastes and extra water from blood. Urine travels from the kidneys through two thin tubes called ureters and fills the bladder.
And Q. 5 order, 3, 2, 1, 4.
Explanation:
Answer:
The structure of the cell is very much linked with the functions of the cell.The cardiac muscle is made of myocardium which serves the body by providing the blood, nutrients and oxygen.The cardiac muscles appears stripped when seen under microscope.
The skin consists of the tissues and cells which are close enough to create a boundary which does not allows the entry of materials and pathogens inside the body.
Hence, the function and the morphology of the cells are somewhere related to each other.
Answer:
Aa
Explanation:
In Punnett Square, you are bringing the letters on the top to the numbered squares, if that makes sense. It is to predict the genotype of offspring.
So square 1, on the left is A, and on the top is A, so it would be AA.
For square 2, on the left is a, and on the top is A, so it would be Aa.
For square 3, on the left is A, and on the top is A, so it is AA.
Square 4, your question, has a on the left, and A on the top, so it is Aa.
Hope it makes sense. You should probably watch a Punnett Square tutorial online or something for a full and more general explanation.
Answer:
The old idea that coronary heart disease is an infectious disease has gained popularity in recent years, and both viral and bacterial pathogens have been proposed to be associated with the inflammatory changes seen in atherosclerosis. Herpes group viruses, notably cytomegalovirus and herpes simplex type 1, have been associated with atherosclerosis and restenosis. Helicobacter pylori and dental infections have also been linked to atherogenesis, but the evidence seems to favor a respiratory, obligatory intracellular bacterium, Chlamydia pneumoniae. The association was originally found in seroepidemiological studies, but the actual presence of the pathogen in atherosclerotic lesions has been repeatedly demonstrated, and during past year the first successful animal experiments and encouraging preliminary intervention studies were published. The causal relationship has not yet been proven, but ongoing large intervention trials and continuing research on pathogenetic mechanisms may lead to the use of antimicrobial agents in the treatment of coronary heart disease in the future.
Explanation:
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