Hallo~~
Your question is related to genetics.
Have you ever done a Punnett Square? This question forces you to make one. If your cross a homozygous type A (AA) and homozygous type O (OO) you should get 100% AO. Understanding genetics, an AO gene means a type A blood. Therefore, it's 100% type A blood (AO).
Answer: One H⁺ ion ie required in converting ATP and inorganic phosphate to ATP
Explanation:During oxidative phosphorylation, high energy electrons released by hydrogen carriers are shuttled through the electron transport chain. The released energy is used to translocate 3 H+ ions from the matrix, creating an proton motive force, which will cause 1 H+ ion to move down the electrochemical gradient and diffuse back into the matrix (chemiosmosis) which is facilitated by ATP synthase. As the H+ moves through the ATP synthase this triggers the molecular rotation of the enzyme, synthesizing ATP
Answer:
DNA template sequence:
5' TGACCAAGT 3'
RNA antisense sequence: 3' UGACCAAGU 5'
>>> RNA sense sequence (transcript) : UGAACCAGU
Explanation:
In the DNA molecule, four types of nitrogenous bases are found: cytosine (C), and guanine (G), adenine (A) and thymine (T). In DNA the bases pair up with each other in the following ways: A pairs with T by two hydrogen bonds, while C pairs with G by three hydrogen bonds. In RNA (i.e., transcript sequence), T is replaced by uracil (U). Moreover, the RNA nucleotide sequence is read in the direction 5' to 3'.
Mitosis should be the answer. if not that, then cell division. hope this helps :3
Answer:
1. Acetylcholine binds to receptors on the motor end plate
2. Ligand-gated channels open leading to depolarization
3. End plate potential triggers an action potential
4. Transverse tubules convey action potentials into the interior of the muscle fiber
5. Calcium is released from the sarcoplasmic reticulum
6. Calcium ions bind to troponin, which then moves tropomyosin
Explanation:
Acetylcholine (ACh) is a signaling molecule (neurotransmitter) that binds to receptors on muscle cells. This binding triggers the opening of ligand-gated sodium channels, thereby ions enter into muscle cells, which causes the depolarization of the sarcolemma and thus promotes the release of Ca2+ ions from the sarcoplasmic reticulum. The myoneural junction, also known as the motor endplate, is the site of synaptic contact between a motor axon and a skeletal muscle fiber. The endplate potential is the voltage that produces the depolarization of muscle fibers when ACh molecules bind to their receptors in the cell membrane. This depolarization spreads in the sarcolemma through transverse tubules (T tubules) and thus generates an action potential. Finally, this action potential induces the release of Ca2+ in the sarcoplasmic reticulum, which activates troponin protein and induces muscle contraction.
