Vaccines are the most efficacious means of minimizing the impact of infectious diseases on the human population. The challenges and importance of making vaccines that will meet FDA approval have never been greater. Genomics has the potential to improve the process of vaccine development substantially. Genome sequencing can help to identify genetic patterns related to the virulence of a disease, as well as genetic factors that contribute to immunity or successful vaccine response. All this information could lead to vaccines with better and more specific targets that elicit more successful protective immune responses. Comparing the genome sequences of viruses that cause infection with those that do not may provide additional insights. In turn, genome manipulation can facilitate derivation of attenuated strains or other vehicles for delivery of the desired antigens to stimulate immune response. On the other end of the spectrum, analysis of host diversity can reveal effective immune responses and possibly the genetic basis for inappropriate response. The recent progress in definition of the innate immune system, necessary for acquired response, should facilitate the definition of this host diversity.
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The main components are the blood cells such as white blood cells, red blood cells, and platelets. The other components represent additional information about these cells including their size, color, function, and maturity.
The pathophysiology of dementia of the Alzheimer type is loss of volume of brain tissue as neurons deteriorate and die.
<h3>What is Alzheimer disease?</h3>
Alzheimer disease is a type of disease that affects the brain cells and it's otherwise called a neurosis.
It is a neurodegenerative disease that is characterized by dementia, that is initial memory impairment and cognitive decline.
Pathophysiology of a disease is the pathway that shows how the disease affects the physiology of the body systems.
The pathophysiology of Alzheimer's disease include:
- The beta-amyloid deposition and neurofibrillary tangles lead to loss of synapses and neurons,
- This results in gross atrophy of the affected areas of the brain leading to death of brain cells.
Therefore, the pathophysiology of dementia of the Alzheimer type occurs due to beta-amyloid deposition which leads to loss of volume of brain tissue.
Learn more about dementia here:
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