Answer: If a mutation occurred in Silencer of Death Domain (SODD) that prohibited its interaction with the DD of TNF receptor, the TNF receptor would evoke its response by binding to a transmembrane receptor, TNFR1.
Explanation: Binding to TNFR1 inhibits the recruitment of cytoplasmic signaling proteins to TNFR1 to prevent recruitment of a number of protein (TRADD) or prevent spontaneous aggregation of the cytoplasmic death domains of TNFR1 molecules.
SODD mechanism is useful in preventing unwarranted TNFR1 activation in the process of apoptosis.
I think the enzyme photolyase is activated by light and can repair thymine dimers caused by UV radiation. Exposure to UV radiation is the main factor that causes skin cells to become cancer cells. These enzymes are involved in the repair of ultraviolet light-induced DNA damage when activated by light absorption.
<span>C. Gametes are the sex cells, and each (sperm and ovum) contain half of the parent's genetic material. These cells will fuse to produce a zygote, which will usually contain the full number of genes required by an organism to live. If there is a chromosomal abnormality, such as a gamete with an extra chromosome, the offspring can either die in development or sometimes be born with physiological and/or developmental difficulties. An example of this phenomenon is trisomy 21, also known as Down Syndrome, in which the 21st chromosome bears 3 copies instead of the regular 2.</span>
<span>Russell's case (two illnesses: bipolar disorder and substance use disorder) illustrates </span>comorbidity. The term comorbity is used in medicine to denote a presence of two diseases or disorders, one primary and one additional, both occurring at the same person.
Answer:
There are two possibilities: The offspring can either be HH or Hh; therefore, there is a 50% chance of the offspring being one of the two above. This means that there is a 50% chance off the offspring being tall or short.