Answer:
its like a cycle, theey go around n around to produce sometime of object or gas
Explanation:
Answer:
The policy for controlling environmental mercury pollution should address ways to prevent and control this pollution. Policy:
- Ban the incineration of waste
- Require that coal-burning companies remove mercury from the coal
- Allocate funds towards research and development for renewable energy resources in the hopes of switching away from coal.
- Require that products containing mercury be labeled as such.
- Set up programs that will recycle batteries and mercury-filled products.
- Set up education programs that will help inform people about mercury pollution.
This policy works by addressing the ways to prevent and control mercury pollution.
Three problems that could result from implementing this policy:
- Backlash from coal-burning companies.
- It could take a while before we completely shift away from using coal.
- Some of the programs that can be set up in this policy can be too expensive to set up and maintain.
A. They do not exhibit contact inhibitors
B. They lack specialization
<span>C. They have abnormal chromosomes </span>
<span>D. They fail to undergo apoptosis</span><span>
A normal, typical and functional cell undergoes cell cycle in normal fashion and eventually reaches apoptosis. Yet cancer cells fail to display just some of these characteristics.
</span><span>The cycle cycle; mitosis occurs more in your body since it changes, modifies and requires cell division at maximum rate in many useful situations with the stand to a particular system and organ. </span>
"REPRODUCTIVE ISOLATION" needs to occur for speciation to happen.....
Answer: The relationship between blood pressure and heart rate responses to coughing was investigated in 10 healthy subjects in three body positions and compared with the circulatory responses to commonly used autonomic function tests: forced breathing, standing up and the Valsalva manoeuvre. 2. We observed a concomitant intra-cough increase in supine heart rate and blood pressure and a sustained post-cough elevation of heart rate in the absence of arterial hypotension. These findings indicate that the sustained increase in heart rate in response to coughing is not caused by arterial hypotension and that these heart rate changes are not under arterial baroreflex control. 3. The maximal change in heart rate in response to coughing (28 +/- 8 beats/min) was comparable with the response to forced breathing (29 +/- 9 beats/min, P greater than 0.4), with a reasonable correlation (r = 0.67, P less than 0.05), and smaller than the change in response to standing up (41 +/- 9 beats/min, P less than 0.01) and to the Valsalva manoeuvre (39 +/- 13 beats/min, P less than 0.01). 4. Quantifying the initial heart rate response to coughing offers no advantage in measuring cardiac acceleratory capacity; standing up and the Valsalva manoeuvre are superior to coughing in evaluating arterial baroreflex cardiovascular function.
Explanation:
