The question is incomplete, the complete question is:
A client with myasthenia gravis improves and is discharged from the hospital. The discharge medications include pyridostigmine bromide (Mestinon) 10 mg every six hours. The nurse evaluates that the drug regimen is understood when the client says, "I should:
1
Take the medication on an empty stomach."
2
Set an alarm so I take the medication on time."
3
Take my pulse rate before taking the medication."
4
Monitor for an increase in blood pressure after taking the medication."
Answer:
The correct answer is statement 2.
Explanation:
Pyridostigmine refers to an essential drug, which must be consumed on time, as a late or missed dose can lead to extreme neuromuscular and respiratory consequences or even death. Pyridostigmine should be consumed with a small concentration of food in order to inhibit gastric irritation.
It is not essential to take the pulse rate prior to taking the medication. Pyridostigmine may result in hypotension, however, not hypertension, which is not an indication of cholinergic crisis.
I think that p<span>igments </span>absorb light<span> used in </span>photosynthesis. In photosynthesis<span>, the sun's </span>energy<span> is converted to chemical </span>energy<span> by photosynthetic organisms. ... For instance, </span>plants <span>appear green to us because they contain many chlorophyll a and b molecules, which reflect green </span>light<span>.
I think this is the answer. </span>
Answer:
Box one: A compound
Box two: bonded chemically
Explanation:
it's a molecule, which is a collection of atoms bonded together chemically through their electrons.
hope that helps!
Answer:
C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.
Explanation:
The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.
In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.
When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.
Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.
