Answer:
The correct answer is option B, that is, promoter, operator, gene.
Explanation:
In a prokaryote, just in front of the operator, the promoter is located, and just in front of the structural genes, the operator is located. All these in combination give rise to an operon.
Any gene, which codes for an enzyme or a structural protein is termed as a structural gene, in the lac operon, a structural gene codes for three distinct kinds of enzymes. A short sequence of DNA is situated just in front of the structural gene, which is known as the operator. An operator functions as a switch for transcription, that is, it monitors whether the process of transcription will take place or not.
Just in front of an operator, a sequence of a promoter is present, and for the transcription and translation of all the structural genes, the RNA polymerase needs to combine first with the promoter. In case, if no transcription is to take place, then the operator allows the repressor to bind with it and prevent the attachment of RNA polymerase with the promoter. Hence, the correct sequence of a prokaryotic operon is a promoter, operator, and gene.
Organisms that cause disease are carriers. Such as ticks, fleas, lice.
Explanation:
A single nucleotide-pair substitution missense mutation causes a change of a single amino acid into another. Aa a result, the produced protein will have an almost normal sequence except for one amino acid.
On the other hand, a frameshift mutation changes the Open Reading Frame (ORF) of the ribosome. The ribosome moves along the mRNA every three nucleotides (codons) and translates them into amino acids that will form the nascent protein. If there is a frameshift mutation (an insertion or deletion of a number of nucleotides not multiple of three) the ribosome will "read" the mRNA differently and will identify different codons than the wild-type sequence, so a large number of amino acids will be different in the mutated protein.
A mutation may result change in the way you look, you could lose sight, could be born with some genetic issues.
Answer:
The plant cell wall is strengthened by the molecular structure of cellulose. Cellulose is made up of ß-glucose arranged upside down, this arrangement aided hydrogen bonds between the hydrogen ions of the hydroxyl group and oxygen of the of the ring of same betta -glucose.
The aggregation of the hydrogen bonds give bundles of strong tensile strength of cellulose called the microfibrils (of 60-70 celluose molecules).They are held together in bundled called fibers.T<u>hese is the source of plant cell walls strength.
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Collagen is the main extracellur matrix (EM) in animal cells.It is a glycoprotein made up of 25%of body protein of animals.Each collagen molecule is made of helix shaped ,three polypeptide chains, wound around each other to form<u> triple helix.</u>The bonds holding helix together are hydrogen and covalent bonds.
Each triple helix is attached to adjacent collagen molecule, parallel to it. The covalent bonds formed a cross link which held the collagen molecules together forming FIBRILS. This gives flexibility to collagen, while maitaing strong tensile strength. This is what is responsible for the structural strength of cell membrane
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.The EM,is futher reinforced with carbohydrate molecules(proteoglycans) which<u> aided in water movements by osmosis following sodium movements into the matrix.</u>