Coprophagy is important for the nutritional balance of rabbits and their relatives.
The behaviors of the rabbit, rat, mouse, hamster, guinea pig and chinchilla have all been linked to coprophagy, which is the act of an animal re-consuming its own discharged excrement. Cecotrophy is a better name for it.
The omnivorous rats, mice, and hamster display cecotrophy to a lesser level than herbivores' hindgut fermenters like the rabbit, guinea pig, and chinchilla. In order to boost the supply of folic acid and vitamin B12, it may rise during pregnancy and lactation.
Coprophagy is an intrinsic behavior triggered by the reflex, despite the fact that modern diets make it unnecessary for survival.
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D. taxonomy
tax = order, classify
nom = name
Answer:
they are Na+, K+ and Ca2+ ion channels.
Explanation:
Ionotropic acetylcholine receptors are also called nicotinic acetylcholine receptors because beside acetylcholine (Ch) they respond to nicotine. These receptors are primary receptors in muscle for motor nerve-muscle communication that controls muscle contraction.
Two molecules of ACh are required for receptor to open. Since the receptors are linked to ion channels, the channels open. Opening of the channel allows positively charged ions to move across it: sodium enters the cell and potassium exits.
Caffeine cause dose dependent prominent increase in force of contraction and period of contraction of muscle
<u>Explanation:</u>
Caffeine is widely used in foods and drinks. Its long time use can effect central nervous system include lethargy memory, sleep disorder and anxiety.
Increase in concentration of calcium ion will result increase number of power stroke and force for contraction. However latent period and relaxation period are unaffected by caffeine. Increase in concentration of calcium ion will result increase number of power stroke and force for contraction.
Because of calcium ion release in skeletal muscle, caffeine causes peripheral manifestation such as hyper-reflexia, muscle twitching etc.
High amount of caffeine leads to increase in release of Ca2+ from sarcoplasmic reticulum. It increases tension development in single tetanic contraction and re-initiate maximum tension in fatigue single myofibre by restoring Ca2+ release from sarcoplasmic reticulum to non-fatigued levels.
If they did not communicate then there wouldn't be a logical explanation for the experiment ..
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