An action potential in an excitatory presynaptic nerve. 2. an _____ in the dendrites of the postsynaptic nerve. 3. this spreads
passively to the ___________. 4. depolarization of this region opens voltage-gated ______ channels. 5. suf?cient membrane depolarization to _________ opens enough of these channels to produce an action potential. 6. in non myelinated axons: the currents associated with the action potential spread to the ______ region of the axon. 7. in myelinated axons: the currents associated with the action potential spread to the next ________. 8. the action potential travels down the axon to the ___________.
An action potential in an excitatory presynaptic nerve. 2. an <u>excitatory postsynaptic potential </u>in the dendrites of the postsynaptic nerve. 3. this spreads passively to the <u>Axon.</u> 4. depolarization of this region opens voltage-gated <u>Na+ and K+ ionic</u> channels. 5. sufficient membrane depolarization to <u>reach action potential threshold</u> opens enough of these channels to produce an action potential. 6. in non myelinated axons: the currents associated with the action potential spread to<u> the depolarized </u>region of the axon. 7. in myelinated axons: the currents associated with the action potential spread to the next <u>Nod of Ranvier</u>. 8. the action potential travels down the axon to the <u>Axon terminals.</u>
An action potential is an excitatory presynaptic nerve. An EPSP (Excitatory postsynaptic potential) takes place in the dendrites of the postsynaptic nerve. This spreads passively to the axon hillock. Depolarization of this region opens voltage-gated ionic channels. Sufficient membrane depolarization to threshold opens enough to these channels to produce an action potential. Non myelinated axon; the currents associated with the action potential spread to the depolarized region of the axon. In myelinated axon; the currents associated with the action potential spread to the next Node of Ranvier. The action potential travels down the axon to the axon terminals.
The cause of the client’s pain must be because of atrophy of
bulbouretheral glands. The bulbourethral glands are the one responsible for
secreting a substance which we call alakaline, in order to be able to provide
neutralization in the acidic environment in a person’s urethra. The
bulbourethral glands are also responsible in having to provide lubrication to a
person when having to do a sexual intercourse. If it gradually declines in its
effectiveness, during sexual intercourse, it would likely cause the person to have
pain and have difficulty in having to do an intimacy with the person’s partner
as atrophy may be present in the person’s bulbourethral gland.