He demonstrated that SCD and sickle cell trait were due to the presence of abnormal 8-globin polypeptides in red blood cells. He demonstrated that the electrophhoretic mobility of B-globin from patients with SCD was different from that of healthy individuals. He demonstrated that both parents of multiple patients with SCD had low levels of sickled red blood cells. He hypothesized that SCD was a recessive trait and that the parents of patients with SCD would be heterozygous carriers. He demonstrated that the difference between B-globin polypeptides in individuals who were healthy and those with SCD is an amino acid substitution. He performed a peptide fingerprint analysis on B-globin from individuals with 84 84 and 89 88, which identified the segment of B-globin that was changed by the BS mutation. James Neel Linus Pauling Vernon Ingram
hope it helps..
Answer:
Due to no transfer of resistance allele.
Explanation:
The gene flow was not the cause of antibiotic resistance because the characteristics can't be transferred from one generation to the next generation. The organisms are evolved to become an antibiotic resistance due to their environmental conditions and this resistance quality is only present in the existing population and the alleles that are responsible for this resistance can't be transferred from the bacteria to the next generation so the experiment showed that gene flow was not the cause of antibiotic resistance
This is a question that you would have to answer
for example: i found a small and colorless bear looking animal(sight). When i touched it was fuzzy and soft(touch). It smelled like dirt and lavender(smell). The creature was sleeping and it was breathing slowly(hearing). I took one of its hair’s and it was like human hair(taste)
BUT REMEMBER since it’s a species it could be an animal or human or a plant.
hopefully I could help!!
When the bond between the second and third phosphate group is broken in ATP, ADP is formed and energy is released. This energy can power the cell's functions.
