Answer:
The team would have to replace the nucleus.
Explanation:
Prokaryotic cells, such as the Mycoplasma capricolum cell used in the experiment do not contain either membrane bound organelles or a defined nucleus. Prokaryotic DNA floats around freely in the cytoplasm in a region called the nucleoid.
The genetic material of eukaryotic cells is protected by a membrane bound nucleus. Therefore, in order to replace an animal cell's DNA, the whole nucleus has to be removed.
Example:
In the process of cloning, the oocyte (egg cell) that receives the nucleus (from somatic cell) of the desired species or individual has to be enucleated i.e. its own nucleus has to be removed. This process is called somatic cell nuclear transfer.
According to the theory of vitalism all the living substances are special and are distinct from the non-living substances, that is, they comprise a unique entity within themselves. This soul or entity distinguishes them from the non-living components.
However, the theory was rejected when urea was taken out successfully from a non-living matter by Wohler, proving the fact that the biological constituents can be extracted from a non-living source. The modern scientists now believe that the living things are just more composite than the non-living substances.
Ti plasmid encoded octopine and nopaline catabolism in Agrobac terium Ti plasmid-encoded genes required by the micro organism for opine catabolism.
The occ and noc areas in octopine and nopaline Ti plasmids, respectively, are accountable for the catabolism of octopine and nopaline in Agrobacterium. The functions are activated within the presence of the opines with the aid of OccR and NocR, related regulatory proteins, and the promoters incorporate commonplace collection motifs.
we have investigated Ti plasmid in heterologous interactions among the regulators and the promoters. previous experiments the usage of all possible heterologous combos of opines, regulators, and promoters in vivo had demonstrated that handiest the aggregate of nopalme, NocR, and the occ promoter led to restricted promoter activation. We now display that OccR and NocR bind to the heterologous promoters in vitro and in vivo.
The weak or non-existent promoter activation truly located can be explained by the idea that OccR and NocR use distinct activation mechanisms; we investigated protein-brought about DNA bending due to reports that the two regulators vary in this respect.
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Answer:
I believe it is D
Explanation:
If an antibody is produced for a bacteria, then it would be specific to that bacteria. If the bacteria evolves, an new antibody would have to be produced to mach that new strain of bacteria. I could be wrong, but that is my reasoning. Hope it helps :)